BCL

Conclusion: Bcl-2 was restraining apoptosis gene, Bax, Bak were promoting apoptosis gene.

结论: Bcl一2为抑凋亡基因, Bax和 Bak为促凋一亡基因, 它们可以作为评估喉鳞癌生物学特性的参考指标.

Conclusion Bcl-2 , Bax, Bcl-xl proteins might participate in the carcinogenesis and development of BCC.

结论Bcl -2 、 Bcl-xl 、 Bax蛋白可能参与了基底细胞癌的发生、发展.

The expressions of bcl-x, bax and eNOS were measured by SQ-RT-PCR technique.

采用半定量逆转录聚合酶链反应(SQ-RT-PCR)法检测内皮细胞bcl-x 、 bax以及 eNOS表达.

Conclusion Bcl-2 gene family may participate in the DATS induced apoptosis of MGC-803 cells.

结论Bcl -2 基因家族可能参与了DATS诱导的MGC -803 细胞凋亡.

Conlusion 1 Over expression of Bcl-2 was observed in biliary carcinomas.

结论1Bcl -2 蛋白在胆管癌与良性胆管疾病中表达有显著差异,它的过度表达与肝外胆管癌的发生有关.

The Study on Effect of Apoptosis-related Gene C-myc, bcl-2 and p 53 in Infancy Hemangiomas.

凋亡相关基因C-myc, bcl -2 ,p53在小儿血管瘤中的作用研究.

So to a certain extent, bcl-2 and bax expression might reflect the DG and biological behavior.

推测bcl -2 、bax表达水平在一定程度上反映了肿瘤的生物学行为,可作为预测预后的指标.

The staining of TOPO-II α, P-gp, GST-π and BCL-2 are no significant different between them.

而TOPO- 、 P-gp 、 GST - π、BCL -2 的表达差异无显著性.

The expression of Fas increased with PYM concentration while the expression of Bcl-2 decreased.

Fas表达随药物浓度增加而递减,Bcl -2 表达随药物浓度增加而升高.

The expression of p 185 was reversely correlated to that of bcl-2 in ChC.

p185表达与bcl -2 表达呈负相关.

Bcl-2 protein immunoreactivity was associated with mitochondria and ER.

Bcl2分布与线粒体和内质网有关.

Conclusion: QY and YP can lower the expression of Bcl-2 but not act on P 53.

结论: 清毒饮、养正片确能下调Bcl -2 表达,而对P53作用不够明显.

Conclusion: Narenmandula-11 plays role in inhibiting tumor, which may be related to upregulation of bcl-2 protein.

结论: 那仁满都拉-11,有抑制肿瘤的作用, 可能与bcl -2 蛋白上调有关.

The lev-els of Bcl-2 and Bcl-X _ L proteins were examined by western blot analysis.

Westernblot法分析细胞内Bcl -2 和Bcl -X _L蛋白水平的变化.