EAMG

Objective To investigate the effect of immature dendritic cells ( iDCs ) on experimental autoimmune myasthenia gravis ( EAMG ).

目的　探讨未成熟树突状细胞 ( iDC ) 对实验性自身免疫性重症肌无力 ( EAMG ) 的影响.

CONCLUSION The human anti-AChR antibodies, secreted from the mice transgenic for human Ig loci, induced with Torpedo AChR, are able to bind to AChR in the mouse muscles, which leads to the damages of AChR, resulting in the animal EAMG.

[结论]电鳐乙酰胆碱受体刺激人免疫球蛋白转基因小鼠产生的人抗乙酰胆碱受体抗体，可与肌肉组织中的乙酰胆碱受体结合，导致肌肉收缩无力，可诱导重症肌无力。

The achievement ratio of EAMG model was 75%. 2.

验证EAMG模型成模率达75%。

Immune Tolerant Treatment of Nasal Mucosa with IFN-γ to EAMG

IFN-γ鼻黏膜免疫耐受治疗EAMG的实验研究

Methods: Normal mice, immunosuppressive mice and EAMG rats were used to investigate the mechanism.

方法：采用正常小鼠、免疫抑制模型小鼠和EAMG大鼠模型观察重肌灵片的免疫调节作用。

RESULTS: Model rats showed the symptoms and signs which imitating EAMG.

结果：模型大鼠表现出类似实验性自身免疫性重症肌无力（EAMG）动物模型样症状。

An evolutionary algorithm based on multi-player game theory ( EAMG) for the TSP was proposed.

提出了一种基于多人博弈的演化优化方法（EAMG），用于解决旅行商问题（TSP）。

Active and passive immunity induced EAMG model was used often.

常用的诱导EAMG模型的方法有主动免疫和被动免疫。