EIAV

The transcription of EIAV was regulated by several viral and cellular factors;

EIAV在繁殖过程中的转录涉及到多种因子的调节，其中TAT蛋白是病毒编码的反式激活因子，是病毒复制必须成分。

The novel method is directly applicable to research on the immune mechanism of the EIAV attenuated vaccine.

该方法的成功建立和应用为研究EIAV减毒疫苗的免疫机制提供了好的研究手段，也为其他病毒的免疫学研究提供了新的参考方法。

DLA-EIAV can be served as the animal model forstudying on humanimmunodeficiency virus ( HIV ).

在这一过程中, EIAV L株 由强毒变为弱毒,用此弱毒疫苗接种动物后不表现临床症状,但可诱导 免疫 保护.

Complete Sequence of Proviral DNA of EIAV Chinese Donkey Leukocyte Attenuated Strain

马传染性贫血病病毒驴白细胞弱毒疫苗株前病毒全基因组核苷酸序列分析

Promotingefficacy of the longterminal repeat ( LTR ) of the DLA-EIAV was charactenzed.

DLA-EIAV 株与其亲本马强毒L株 和驴强毒DA-EIAV 株核苷酸序列相比,同源率分别为97.0%和97.5%.

The results also demonstrated that insertion of short sequence in S2 gene does not affect in vitro replication of EIAV in target cells.

本研究还证明了S2基因中的插入突变并不影响马传染性贫血病毒的体外复制。