LPS

In this reaction, PS was decomposed into polysilane of low molecu-lar weight ( LPS ) containing Si-C skeleton and Si-H bonds.

在这一反应中, PS裂解为含Si—H键的低分子聚硅烷 ( LPS ).

Conclusion LPS can inhibit the apoptosis of PMNs.

结论LPS对PMNs的凋亡有抑制作用.

Conclusion LPS could involve bone tissue in inflammation and inhibit the differentiation of osteoblast.

结论LPS刺激骨组织发生炎症反应、抑制成骨细胞分化.

Conclusion LPS can stimulate expression of ESM 1 in endothelial cells.

结论　LPS对ECV304中ESM1的表达有促进作用.

At the end of 1998, there were 85 ODPs and 336 LPs.

迄一九九八年底, 已拟备的发展大纲图有85肺垃发展蓝图则有336份.

The ultrastructure of Clara cells damaged significantly 24 h post LPS injection.

LPS致伤后24h,Clara细胞的超微结构明显受损.

In addition , apoptosis did occur in AEC isolated from TNF KO mice following LPS stimulation.

在TNF基因剔除(knockout,KO)小鼠,LPS仍 可明显诱导AEC凋亡.

CONCLUSION: LPS induces liver IL-6 expression , which might activate BEC proliferation through STAT 3 pathway.

结论: LPS刺激导致肝组织IL-6分泌增加, 后者可能通过STAT3介导BEC增殖.

Notably, treating mice with GSK-3 inhibitor showed a decrease in LPS-induced renal cell apoptosis in vivo.

除了抗发炎作用, GSK -3 抑制剂可以降低体内LPS诱发小鼠肾脏组织中的细胞凋亡现象.

NF-κ B inhibitor CAPE suppressed LPS-induced these effects.

NFκB抑制剂咖啡酸苯乙酯(CAPE)抑制了LPS介导的上述效应.

ACP, GOT, GPT, CHE, CK, LPS and LDH activities had little correlation with fiber digestibility and Met, Cys digestibility ( P & gt; 0.05 ).

酸性磷酸酶(ACP) 、 谷草转氨酶(GOT) 、 谷丙转氨酶(GPT) 、 胆碱酯酶(CHE) 、 肌酸激酶(CK) 、 脂肪激酶(LPS)和乳酸脱氢酶(LDH)的活性与营养物的消化率呈弱相关 ( P & gt; 0.05 ).

MAP , PaO _ 2 , pH and serum TNF-α concentrations were recor ded every hour after LPS.

5h内观察动物的MAP、PaO2 、 pH,血清TNF - α.

Conclusion: The role of LPS in caries-induced reparative dentin might be a significant determinant.

结论: 提示适度的LPS在牙髓对龋病的防御反应过程中起着一定调节作用.

PurposeTo study extraction, purification and physicochemical analysis of Laminaria polysaccharide sulfate ( LPS ).

目的研究海带硫酸多糖 ( LPS ) 的提取制备及其组分的纯化和理化性质.

There were 76 ODPs and 285 LPs covering the HKSAR.

年底时,全港共有76份发展大纲图和285份发展蓝图.