glomerular

DNA fragmentation localized to glomerular cells.

定位于肾小球细胞的DNA断裂.

Results Among 42 cases electrophoresis indicated 27 glomerular proteinuria; 15 mixed proteinuria.

结果42例中27例为肾小球性蛋白尿, 15例为混合性蛋白尿.

The branches reach to glomerular basal membrane.

分支机构达到肾小球基底膜.

Results consistently demonstrated that DNA colocalized with autoantibodies in glomerular membrane associated EDS.

结果始终表明,DNA与肾小球膜电子致密带的自身抗体的共区域.

Objective : To investigate the mechanism inducing apptosis of human glomerular mesangial cells ( Mcs ) by anti Fas antibody.

目的: 研究抗Fas抗体 对人肾小球系膜细胞的致凋亡作用,探讨Fas? FasL 在肾脏损伤中的作用.

Glomerular changes are both frequent and severe.

肾小体的病变是常见而严重的.

The glomerular filtration rate remains relatively constant.

肾小球滤过率也能保持恒定.

Results In normal kidney tissues, ILK was mainly located in the glomerular visceral epithelial cells.

结果在正常组肾组织, ilk主要表达于肾小球脏层上皮细胞.

Furthermore, glomerular damaged showed a significant correlation with tubular DNA damage in proliferatie GN.

另外, 在增生性肾小球肾炎中损伤的肾小球和肾小管DNA断裂显著相关.

Methods Changes relating to glomerular function, structure and glomerular basement membrane ultrastructure were observed.

方法观察低分子肝素对肾小球功能 、 形态及肾小球基底膜超微结构的影响.

Results In normal kidney tissues, ILK was mainly located in the glomerular visceral epithelialcells.

结果在正常组肾组织, ILK主要表达于肾小球脏层上皮细胞.

Cast preparation of the glomerular vessels ( glomerulus ) in a renal corpuscle.

铸造编制肾小球船只 ( 肾小球 ) 在肾小体.

The development of severs hypertention in glomerular disease is an ominous s ign.

肾小球疾病时发生严重高血压是一种不良的现象.

Immune complexes nephritis is a typical nephritis model, its pathological change finally lead to glomerular sclerosis.

免疫复合物型肾炎是一种经典的肾炎模型, 其病理改变最终将导致肾小球硬化.

Magnified portion of a section showing the glomerular capillaries and Bowman's space.

放大部分的一节显示肾小球毛细血管和鲍曼的空间.