Objective To investigate cardiomyocyte apoptosis in viral myocarditis ( VMC ) mice and its relationship with VMC.

Posttraumatic myocarditis usually occurs several days after injury.

受伤数日后,通常发生伤后心肌炎.

This paper summarized 4 cases of nursing acute severe myocarditis similar to acute myocardial infarct.

总结4例酷似心肌梗死的急性重症心肌炎患者的护理.

Immune mechanism is very important to develop myocarditis.

免疫机制在心肌炎发病机制中有着重要的作用.

Assessing the Myocardium After Attempted Reperfusion: Should We Bother?

试图再灌注后评估心肌: 我们应否打扰?

Metaboilic level of myocardium is an important factor.

心肌本身的代谢水平是最重要因素.

Table 2 . Contents of Mb, myocardium protein and LDH activity ( ± s ).

表 2. 实验各组冠状动脉灌洗液中肌红蛋白 、 肌蛋白含量和乳酸脱氢酶活性.

Changes of myocardium blood can markedly alter IVC motion.

结论:心肌供血状态的改变可以引起等容收缩波显著变化.

The metastatic melanoma is seen here to be infiltrating into the myocardium.

图示转移性黑色素瘤浸润到心肌.

AIM: To define the dynamics of energy metabolism in stunned myocardium.

目的: 观察顿抑心肌状态时心肌能量代谢变化规律.

Here is a microabscess in the myocardium.

图示心肌微脓疡.

The patient demonstrates changes in personality, behavior, and memory, followed by myoclonic jerks, blindness, and spasticity.

病人先表现为人格, 行为和记忆力的变化, 然后出现痉挛性的反射, 失明及强直状态.

Objective To study the clinical and EEG characters of the juvenile myoclonic epilepsy.

目的分析青少年肌阵挛性癫痫的临床及脑电图特点.

Method Ion currents of rabbit ventricular myocyte were recorded using the whole cell patch clamp technique.

方法应用全细胞膜片钳技术记录兔心室肌单个细胞的电流.

This article reviews the main mechanisms of preventing cardiac myocyte hypertrophy via small GTP-binding proteins pathways.

现主要从他汀类药物影响小G蛋白 信号通路方面,阐述其抑制心肌细胞肥大的主要机制.

The characteristics of cardiac myocyte auto-beating without external stimulation make the research sense.

心肌细胞在不受外力作用的情况下具有自主搏动特性,使得对于它的动态分析更有意义.

All cardiac myocyte are capable of spontaneousdepolarization and repolarization of their membrane.

所有的心肌细胞都能自律性的去极化和复极化.

Objective : To investigate whether differences exist between myoepithelial cells of human beings and experimental animals.

目的: 了解人与不同实验动物涎腺中肌上皮细胞的差异.

Objective To investigate the intervention neurotrophic factors ( CNTF ) on weightlessness-induced muscle atrophy and its myofiber phenotype transition.

目的探讨 CNTF 对失重性肌萎缩及其纤维表型转换的干预作用.

And there were endoplasmatic reticulum enlargement and the myofibril contracture in the 4℃ group.

且在4℃组观察到心肌纤维挛缩,线粒体嵴的破坏,内质网扩张,冠状血管挛缩等现象.

Fig . 9 Myocardium cell of Procambarus clark ii . Showing striation of myofibril.

图9螯虾心肌细胞示肌原纤维的横纹.