In sports endurance test, the sample could significantly increased weight-carrying swimming time, inhibited hepatic glycogen consuming during swimming and decreased blood lactic acid level after swimming.

Methods KM mice were used. Their body weight, serum urea nitrogen, hepatic glycogen, swimming-sustaining time of mice with a load, plasma lactic acid content, survival time under anoxia or sodium nitrite poisoning were measured.

方法通过对小鼠体质量值、血清尿素氮、肝糖原、乳酸含量、小鼠负重游泳时间、缺氧生存时间、亚硝酸钠中毒存活时间的测定，了解小鼠体能变化情况。

The hepatic glycogen in test group was more than control group ( P < 0.05) in middle dose group, P < 0.01) in high dose group);

各剂量组小鼠运动后血乳酸含量均低于对照组，高剂量组与对照组相比具极显著差异（P<0.01）；

Determination of hepatocyte apoptosis, hepatic glycogen and collagen fiber Liver tissues were obtained after rats in each group were sacrificed to make the paraffin section.

肝组织细胞凋亡、胶原纤维、糖原检测各组大鼠放血处死后取肝组织，制作石蜡切片。

Content Determination on Hepatic Glycogen of Animal Model on High Glucose and Fat by Oxidase Method

应用氧化酶法对高糖高脂动物模型肝糖原含量的测定

Circadian variation of ginseng total saponins on plasma corticosterone and hepatic glycogen in rats

人参总皂甙对大鼠血浆皮质酮及肝糖原作用的昼夜节律

Also, with the transformation of hepatic glycogen into muscular glycogen, there was an increase in muscular glycogen after exercise.

强力宝能提高小鼠肝糖元含量，促进运动后肝糖元恢复，并通过肝糖元向肌糖元的转化，使运动后肌糖元的含量与恢复亦有所提高。

Hepatic glycogen content in rats fed with high-fat diet was significantly decreased, confirming insulin resistance.

蒽酮法的检测结果显示高脂饲料喂养大鼠肝组织糖原含量显著降低，表明肝组织发生了胰岛素抵抗。

The hypoxic tolerant time, content change of hepatic glycogen and serum urea nitrogen were determined through the weight loading swimming experiment.

通过小鼠负重游泳实验测定小鼠耐缺氧时间、小鼠肝糖原含量改变及小鼠血清尿素氮含量变化等。

The content of hepatic glycogen was decreased ( P < 0.01) in DM mice and increased ( P < 0.01) after APS treatment;

DM组肝糖原含量减少，且显著低于DA组（P<0.01）；

Objective: To investigate the effect of hepatic glycogen content on liver ischemia-reperfusion and its mechanism.

目的：探讨不同水平肝糖原含量在肝热缺血再灌注中的拮抗作用及相关机制。

Conclusion The DNA analysis is helpful for confirmation of the diagnosis of glycogen storage disease Ia and 727G → T in exon 5 may be a prevalent mutation causing glycogen storage disease I a in Chinese.

结论糖原累积病Ⅰa型可直接进行DNA分析以明确诊断，葡萄糖-6-磷酸酶外显子5的727G→T突变的发生在中国人中的发生率有待进一步研究。

Clinical and genomic study of the infant with glycogen storage disease type ⅱ

糖原累积病Ⅱ型的临床分析和基因学检测

Objective To investigate the molecular genetic mechanism of a patient with glycogen storage disease la.

目的研究1例糖原累积病Ⅰa型患者发病的分子遗传学机制。

Advances in the Study of Glycogen Storage Disease Type ⅱ

Ⅱ型糖原累积病的研究进展

Neonatal type IV glycogen storage disease associated with null mutations in glycogen branching enzyme 1

糖原分枝酶1无效突变相关性IV型糖原贮积病新生儿亚型

Renal complications of hepatic glycogen storage disease

肝糖原贮积症肾脏并发症的临床观察

Mutation analysis of glycogen debrancher enzyme gene in five Chinese patients with glycogen storage disease type ⅲ

糖原贮积症Ⅲ型基因突变的初步研究

Objective To develop and evaluate a simple, fast and accurate prenatal diagnosis method for glycogen storage disease Ia ( GSD Ia) in Chinese.

目的探讨中国人Ia型糖原累积病简便、快速、准确的产前诊断方法。

Research Advances of Glycogen Storage Disease Type I

糖原累积病Ⅰ型研究的新进展

Therefore, physicians must have sufficient recognition for this disease, and be on the alert for hepatic glycogen storage disease, give a right and prompt diagnosis combined with family history, physical examinations and laboratory findings.

结论肝糖原累积病临床表现多样，要及时准确地诊断本病，需要对本病有充分的认识，结合患者的家族史、临床表现及各种实验室，影像学检查结果正确地加以综合分析。