Conclusions-Cardiac-specific overexpression of A 20 improves cardiac and inhibits cardiac and fibrosis after acute myocardial infarction.

  • 结论:心脏特异型过表达A20改善心脏功能,抑制急型心肌梗死后的心脏重建、凋亡、炎症和间质纤维化.

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