hepatocirrhosis的例句大全

ADA obviously increases in nearly 90 % cases of hepatocirrhosis.

肝硬化时,ADA有近90%的病例明显上升.

It is concluded that the cause of death in this case was botulism rather than hepatocirrhosis.

本病例死因应为细菌性食物中毒而非肝硬化.

Village doctor, 59. Died of hepatocirrhosis caused by Schistosomiasis.

乡村医生, 59岁,死于由血吸虫病引发的肝硬化.

Objective : To explore the efficacy of astragalus injection and JI SI solution in treating hepatocirrhosis.

目的: 探讨黄芪注射液与激饲液治疗肝硬化的疗效.

Objective : To study the etiology and prevention measures of digestive tract hemorrhage in hepatocirrhosis patients.

目的: 探讨肝硬化并消化道出血病因及预防.

Conclusion It is an ideal operation method in the treatment of hepatocirrhosis and portal hypertension.

结论脾大部切除脾大网膜腹膜后固定术是治疗肝硬化门脉高压症的一种理想术式.

Malnutrition has high occurrence in patients of hepatocirrhosis, which is mainly about protein-energy malnutrition.

肝硬化患者营养不良发生率较高, 以蛋白质 - 能量营养不良为主.

Methods One case of AIH with hepatocirrhosis was treated by orthotopic liver transplantation.

方法对1例AIH肝硬化患者行肝移植手术.

Objective To investigate the diagnostic value of serum carbohydrate antigen 125 ( CA 125 ) in liver carcinoma and hepatocirrhosis.

目的探讨血清 CA125 在肝硬化与肝癌诊断中的价值.

Methods: 91 patients with hepatocirrhosis were classified with Child-Pugh grades and the grades were compared with 4 parameters of thrombocyte.

方法：对91例肝硬化患者进行Child-Pugh分级，并与血小板各项参数进行比较。

Conclusion: The thrombocyte parameters are important indices for evaluating the damage degrees of liver in the patients with hepatocirrhosis.

结论：血小板各项参数是评估肝硬化患者肝功能损害程度的重要指标。

So VEGF is an important reason during anamorphism of hepatocirrhosis.

这说明，在肝硬化渐变中，VEGF起着重要作用。

To study the effect of Qianggan Ruanjian decoction ( QRD) on hepatocirrhosis portal hypertension and its possible function mechanism.

探讨强肝软坚方对肝硬化门脉高压症的作用及其可能机制。

Detection and Clinical Significance of Plasma Diamine Oxidase and Endotoxin in Hepatocirrhosis Patients

肝硬化患者血浆二胺氧化酶及内毒素检测的临床意义

Methods A model of hepatocirrhosis was made with CCL 4.The models were divided into model and electroacupuncture groups and a normal group was established as a control to observe hepatic, serous and intestinal glutathione ( GSH) contents and to quantitatively analyze the state of enteric mucosa.

方法用CCL4将模型组、电针组造成肝硬化模型，并与正常对照，观察肝组织、血清、肠道谷胱甘肽（GSH）含量并定量分析小肠黏膜状态。

Conclusion The level of GADA and ICA in the hepatocirrhosis patients get close to that of LADA indicating that the mechanism of autoimmune injure in the insulin cell are similar to that of LADA.

结论肝硬化患者血清GADA和ICA阳性率与LADA非常接近，说明肝硬化患者存在着与LADA发病机理相似的胰岛细胞自身免疫性损伤，而且随着肝功能下降血清ICA阳性率进行性上升。

Joined treatment of bypath and disconnection for hepatocirrhosis with portal hypertension bleeding

分流断流联合术治疗肝硬化门脉高压症出血

With the higher levels of IL-6 and IL-8, it may promote chronicity of hepatitis and occurring on hepatocirrhosis, and worsened process of hepatitis.

IL-6和IL-8高水平的存在，可促进肝炎的慢性化和纤维化，加重肝炎的病程。