codon

Introduction: About 30 % of inherited genetic disorders in humans result from premature termination codon mutations.

前言: 约30%的人类遗传性疾病起源于过早出现的终止密码子突变.

Because of this coding relationship, triplet of bases is called a codon.

由于这种编码关系, 碱基的三联体被称为密码子.

Furthermore , a fragment of 150 bp from start codon ATG to 150 nt showed the most variation.

从ATG至第150bp区段的核苷酸序列变异频率最高.

We further explored the correlation between synonymous codon bias and gene expression level and gene length.

进一步研究基因表达水平和基因长度与密码子使用偏爱之间的关系.

As a stop codon reaches the A-site of a translating ribosome , either termination or readthrough occurs.

当终止密码子进入核糖体翻译的A位时,将会发生翻译终止或是通读.

CONCLUSION: The expression of LZP 3 gene in yeast could be successfully improved by using codon optimization.

结论: 通过密码子优化,能显著提高LZP3基因在酵母细胞中的表达水平.

Correspondence analysis has frequently been used for codon usage studies.

对应分析法在密码子用法的研究中频繁使用.

However, stop codon susceptibility to gentamicin between different genes is difficult to predict.

但是, 不同基因的终止密码子对庆大霉素的易感性是难以预测的.