ischemia的例句大全

Focal cerebral ischemia was induced by middle cerebral arterial occlusion ( MCAo).

以线栓法制备左侧大脑中动脉阻塞（MCAo），诱导大鼠局灶性脑缺血模型。

Conclusion The modified MCAO model is a cerebral ischemia-reperfusion model in rats with favorable stability and high success rate.

结论改良的大鼠MCAO模型是一种稳定性好、成功率高的脑缺血-再灌注模型。

Methods: A rat model of focal cerebral ischemia was made by MCAO of electric coagulation.

方法：电凝阻断大脑中动脉法制备大鼠局灶性脑缺血模型。

The rat MCAO ischemia/ reperfusion model was made by the intraluminal suture method.

采用线栓法阻断大鼠大脑中动脉产生局灶性脑缺血再灌注模型；

Conclusion Ischemia stroke sequela model of rats can be established in 5 weeks after MCAO.

结论：大鼠MCAO5周后为缺血性中风后遗症大鼠模型。

METHODS: The rats were randomized into 4 groups: ① The control without ischemia ( 10 rats): Vertebral artery was ligatured bilaterally and common carotid artery was not clipped bilaterally.

方法：大鼠随机分为四组：①非缺血对照组（10只）：仅结扎双侧椎动脉而不夹闭双侧颈总动脉。

Effect on Therapy and Expression of Caspase-3 of Baicalin in Rats with Cerebral Ischemia

黄芩苷对脑缺血大鼠的治疗作用及对Caspase-3的影响

BrdU-labelled NSCs were transplanted into the penumbra zone 3 days after cerbral ischemia. The Neurological Severity Score ( NSS) tests were undertaken in all the 3 groups 2-12 weeks after transplantation.

移植后2～12周以神经损害严重程度评分（NSS）评价各组动物神经功能状况。

Objective: To evaluate whether supplementation of folic acid, vitamin B 6 and vitamin B 12 decreases plasma homocystine ( Hcy) level in patients with essential hypertension and ischemia stroke.

目的：探讨叶酸、维生素B6、腺苷辅酶维生素B12对原发性高血压伴缺血性脑卒中患者血浆同型半胱氨酸（Hcy）水平的影响。

Conclusion Ani have a remarkable protection on cerebral ischemia reperfusion damage. Its mechanism may be related with decreasing the neuronal apoptosis.

结论Ani可减少脑缺血再灌注神经元凋亡，提高神经元的存活数目，对脑缺血再灌注损伤起保护作用。

AIM: To study the protective effect of anisodine ( Ani) on acute forebrain ischemia-reperfusion injury in rats.

目的：研究樟柳碱对大鼠急性脑缺血及再灌注损伤的影响。

The analysis of Q TC interval changes in treadmill exercise induced myocardial ischemia

平板运动试验诱发心肌缺血Q-Tc的变化分析

Promoting effect of db-cAMP on axon regeneration and motor function recovery in brain of ischemia-reperfusion rats

双丁酸环腺苷酸促进脑缺血再灌注大鼠轴突再生和运动功能恢复

The research of the mechanism involves in biochemistry, molecular biology and blood dyscrasia, but it's center still is high blood sugar which causes a series changes of ischemia and hypoxia.

发病机制的研究涉及生物化学、分子生物学、血液异常的等方面，但其中心环节是高血糖和其引起组织缺血缺氧发生的一系列改变。

Change of Apoptosis Controlling Gene in Cerebral Ischemia Reperfusion Injury and Its Significance

脑缺血再灌注损伤中凋亡调控基因表达的变化及意义

The coronary ischemia threshold reached minimum at the 10th minute.

冠脉缺血阈值与上述指标相反，10min时为最低。

Hepatic ischemia reperfusion injury and protective effects with infusion of ATP MgCl 2 and oxygenated crystalloid solution and the observation of hepatocellular pathologic changes

肝缺血再灌注损伤和ATP-MgCl2含氧晶体灌注液的保护作用&肝细胞病理变化观察

The frequency of ischemia attacking, paroxysmal cumulative time, motion related incidence were lower in the treated group after treatment than those in the control group.

治疗组治疗后心肌缺血发作频率、发作累计时间、与活动有关发生率均明显低于对照组；

The positive criteria of treadmill exercise testing ( TET) mainly reflect the changes induced by myocardial ischemia in biochemical, electrophysiological and hemodynamic three aspects.

平板运动试验（TET）的阳性标准，虽有不少，但均反映心肌缺血引起的生化、电生理及心肌收缩力3个方面的变化。

Acute incomplete cerebral ischemia was induced by ligaturing bilateral carotid arterise;

小鼠双侧颈总动脉结扎引起急性不完全脑缺血模型，测定小鼠死亡时间；