CONCLUSION: FK506 could induce a significant impairment of intestinal mucosa morphologically, which might be associated with up-regulated expression of iNOS and TNF-α in small intestinal mucosa.

Objective: To examine effects of n-butanol fraction from Fructus Kochiae ( NBFK) on gastric emptying of mice, α-glucosidase activities in rat intestinal mucosa, and glucose absorption in rat intestine as to elucidate the hypoglycemic mechanism.

目的：观察地肤子正丁醇部分（NBFK）对小鼠胃排空、大鼠小肠黏膜α葡萄糖苷酶活性及小肠糖吸收的影响，研究其降糖作用机制。

Objective To investigate the feasibility and benefits of co-culture of cryopreserved islets with small intestinal submucosa ( SIS).

目的探讨猪小肠黏膜下层（SIS）对冷冻保存后大鼠胰岛细胞的支持和保护作用。

Experimental Research of Porcine Small Intestinal Submucosa ( SIS) to Become Material of Lamellar Keratoplasty

猪小肠黏膜下层（SIS）作为板层角膜移植材料的实验研究

Methods A model of hepatocirrhosis was made with CCL 4.The models were divided into model and electroacupuncture groups and a normal group was established as a control to observe hepatic, serous and intestinal glutathione ( GSH) contents and to quantitatively analyze the state of enteric mucosa.

方法用CCL4将模型组、电针组造成肝硬化模型，并与正常对照，观察肝组织、血清、肠道谷胱甘肽（GSH）含量并定量分析小肠黏膜状态。

Conclusion Melatonin can protect small intestinal mucosa barrier through decreasing the shift of intestinal bacteria endotoxin, lowering cell factor level in plasma and inhibiting ICAM-1 expression in intestinal tissue after obstructive jaundice.

结论褪黑素通过减少肠道细菌内毒素的移位，降低血浆中细胞因子水平，抑制梗阻性黄疸后肠组织ICAM-1的表达，保护小肠黏膜屏障。

In vivo tissue engineering& the application of small intestinal submucosa

体内组织工程技术&小肠黏膜下层的应用

Objective To repair rat acute spinal cord injury by using the porcine small intestinal submucosa ( SIS) compound of Schwann cells.

目的应用复合雪旺细胞（SC）的小肠黏膜下层（SIS）修复大鼠急性脊髓损伤。

Objective To review the research advance of the preparation and characteristics of small intestinal submucosa ( SIS).

目的追溯近年有关小肠黏膜下层（SIS）的制备及其特性的研究与进展。

Results: Severe lesions in the jejunum and ileum were caused by indomethacin and the NO 、 MPO and MDA contents were markedly increased ( P < 0.05).

结果：消炎痛引起明显的小肠黏膜损伤，伴有NO、MPO、MDA含量增加及SOD活性下降（P<0.05，P<0.01）。

The third toe of the left and right feet was chosen from 20 chicken respectively of autologous transplantation group and porcine small intestinal submucosa group, flexor digitorum profundus muscle tendon was cut off at middle phalanx, and 2 cm tendon defect model was created.

取自体移植组20只和猪小肠黏膜下层移植组20只鸡的左、右足第3趾，于中节趾骨处切断趾深屈肌腱并造成2cm的肌腱缺损模型。

The Peyer's patches, mesenteric lymph node, intestinal mucosal epithelial tissues were prepared for the lymphocytes suspension and the changes of CD4+ and CD8+ T cells in these sites were determined by SABC immunohistochemistry;

分离Peyer's patches（PP结）、肠系膜淋巴结（MLN）、小肠黏膜上皮制备淋巴细胞悬液并涂片，用免疫细胞化学法检测CD4~+、CD8~+T细胞亚群；

After 105 days experimental observation, the restore of corneal transparency was satisfied on the whole.

将自制的猪小肠黏膜下层（SIS）移植于家兔不同深度角膜植床，经105天实验观察，角膜透明度的恢复基本令人满意。

Effects of Glutamine and Glycyl-Glutamine on Intestinal Mucous in Weaned Piglets

谷氨酰胺和甘氨酰-谷氨酰胺对断奶仔猪小肠黏膜的影响

PURPOSE: Using co-culturing of bone marrow stromal cells ( BMSCs) and small intestinal submucosa ( SIS), to evaluate the cellular compatibility and the osteogenesis ability as well as the feasibility of SIS to serve as a scaffold in tissue engineering.

目的：检测骨髓基质干细胞（BMSCs）与小肠黏膜下层（SIS）复合培养的生物相容性及成骨能力。

The mucosal thickness and the height of villi were diminished after 1 hour of shock, but there was no obvious change in the colon.

小肠黏膜厚度和绒毛高度1h后逐渐减少，大肠各组间则无明显变化。

Conclusion EGF can preserve the mucosal structure of the graft, maintain the integrity of the ultrastructure of graft enterocyte after small bowel transplantation in rat.

结论EGF能较好保护大鼠移植小肠黏膜结构，维持移植肠上皮细胞超微结构的完整。

The respiratory control rate ( RCR), the activity of NADH dehydrogenases and succinate dehydrogenases and cytochrome C were measured to evaluate the function of respiratory chain of the mitochondria in intestinal mucosa.

分离小肠黏膜上皮细胞内线粒体，监测呼吸控制率（RCR）、琥珀酸脱氢酶和烟酰胺腺嘌呤二核苷酸（NADH）脱氢酶活力和细胞色素C的含量。

A hormone released by the upper intestinal mucosa that inhibits gastric motility and secretion.

体积过大;由于过量分泌荷尔蒙所至.

And those who took insulin-stimulating drugs were 2.55 times more likely to develop pancreatic cancer.

而那些服用胰岛素刺激药物的人患胰腺癌的几率提高了2.55成.

Objective To study the immunofunctional changes of cells in different period of pancreas carcinoma.

目的探讨胰腺癌不同时期细胞免疫功能的变化.